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Nanomedicine that targets ATP synthase reverses bone aging in mice

Original reporting: An energy metabolism-engaged nanomedicine maintains mitochondrial homeostasis to alleviate cellular ageing.

On the frontier: Regeneration, Quantum biology

A new class of energy-metabolism-engaged nanomedicines (EM-eNMs) infiltrates mitochondria, triggers fission and mitophagy, and restores stemness in aged bone marrow cells, reversing osteoporotic bone loss in mice.

Three things to remember

  • EM-eNMs bind ATP synthase, inducing DRP1-mediated mitochondrial fission.
  • Systemic delivery selectively targets bone, reversing osteoporosis in aged mice.
  • Mechanism parallels caloric restriction, activating mitophagy to clear damaged mitochondria.

Source

This signal is based on An energy metabolism-engaged nanomedicine maintains mitochondrial homeostasis to alleviate cellular ageing. from PubMed. Read the original report for full context.

Health note: Preclinical study in mice; human efficacy and safety unproven.